This is a Validated Antibody Database (VAD) review about anti-mouse IgG1 secondary antibody, based on 9 published articles (read how Labome selects the articles), using anti-mouse IgG1 secondary antibody product in experiments. It is aimed to help Labome visitors find the most suited anti-mouse IgG1 secondary antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
eBioscience Alexa Fluor 647-mouse IgG1 was used to perform flow cytometry in order to show that IL-17 immunodeficiency could lead to CMCD to the paper
InvitrogenInvitrogen anti-mouse IgG1 secondary antibody product
Invitrogen Alexa Fluor 488 goat antimouse IgG1 was used to perform immunohistochemistry in order to show the architecture of the brain fiber pathways to the paper
Molecular Probes Alexa Fluor 594 goat anti-mouse IgG1 was used to perform immunofluorescence in order to reveal VZV IE63 protein interacts with human ASF1 protein and increases the binding of ASF1 to histone H3.1 and H3.3 to the paper
SouthernBiotech
Southern Biotech HRP conjugated anti-mouse IgG1 was used to perform ELISA in order to show that at late stages of chronic viral infection interleukin-6 is essential for viral control to the paper
BD Biosciences
BD Biosciences FITC-mouse IgG1 was used to perform flow cytometry in order to show that IL-17 immunodeficiency could lead to CMCD 555748to the paper
BD Biosciences anti-mouse IgG1-Al647 antibody was used to perform flow cytometry in order to investigate the importance of phosphatase activity in B cell selection in germinal centers to the paper
Becton-Dickinson rat anti-mouse IgG1-APC was used at 0.4ug/mL to perform flow cytometry in order to show that internal stochastic competition could determine the fates of B cells to the paper
PharMingen rat monoclonal anti-mouse IgG1 was used to perform antibody response analysis in order to show that physicochemical damage could activate T helper 2 cell to induce atopic response through NKG2D to the paper
BD Biosciences FITC-conjugated rat anti-mouse IgG1 was used to perform flow cytometry in order to show that immune activation induced by agonistic CD40 antibodies could be enhanced by inhibitory Fcgamma receptor Fcgamma RIIB engagement to the paper
Bethyl
Bethyl laboratories goat anti-mouse IgG1 antibody was used to detect the reactions by ELISA in order to study the role of inflammasome recognition in adaptive immunity to influenza virus to the paper
Articles Reviewed
  1. Khalil A, Cambier J, Shlomchik M. B cell receptor signal transduction in the GC is short-circuited by high phosphatase activity. Science. 2012;336:1178-81 pubmed publisher
  2. Wedeen V, Rosene D, Wang R, Dai G, Mortazavi F, Hagmann P, et al. The geometric structure of the brain fiber pathways. Science. 2012;335:1628-34 pubmed publisher
  3. Duffy K, Wellard C, Markham J, Zhou J, Holmberg R, Hawkins E, et al. Activation-induced B cell fates are selected by intracellular stochastic competition. Science. 2012;335:338-41 pubmed publisher
  4. Strid J, Sobolev O, Zafirova B, Polic B, Hayday A. The intraepithelial T cell response to NKG2D-ligands links lymphoid stress surveillance to atopy. Science. 2011;334:1293-7 pubmed publisher
  5. Harker J, Lewis G, Mack L, Zuniga E. Late interleukin-6 escalates T follicular helper cell responses and controls a chronic viral infection. Science. 2011;334:825-9 pubmed publisher
  6. Li F, Ravetch J. Inhibitory Fc? receptor engagement drives adjuvant and anti-tumor activities of agonistic CD40 antibodies. Science. 2011;333:1030-4 pubmed publisher
  7. Puel A, Cypowyj S, Bustamante J, Wright J, Liu L, Lim H, et al. Chronic mucocutaneous candidiasis in humans with inborn errors of interleukin-17 immunity. Science. 2011;332:65-8 pubmed publisher
  8. Ichinohe T, Lee H, Ogura Y, Flavell R, Iwasaki A. Inflammasome recognition of influenza virus is essential for adaptive immune responses. J Exp Med. 2009;206:79-87 pubmed publisher
  9. Ambagala A, Bosma T, Ali M, Poustovoitov M, Chen J, Gershon M, et al. Varicella-zoster virus immediate-early 63 protein interacts with human antisilencing function 1 protein and alters its ability to bind histones h3.1 and h3.3. J Virol. 2009;83:200-9 pubmed publisher