This is a Validated Antibody Database (VAD) review about pig APP, based on 45 published articles (read how Labome selects the articles), using APP antibody in all methods. It is aimed to help Labome visitors find the most suited APP antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
APP synonym: amyloid-beta A4 protein; amyloid beta A4 protein; ABPP; alzheimer disease amyloid A4 protein homolog; amyloid beta (A4) precursor protein (peptidase nexin-II, Alzheimer disease); amyloid precursor protein; beta-amyloid precursor protein

Invitrogen
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; pig; 1:250; loading ...; fig 1a
In order to characterize gyrencephalic swine brains exposed to true open field blasts, Invitrogen APP antibody (Life Technologies, 51-2700) was used in immunohistochemistry - frozen section on pig samples at 1:250 (fig 1a). PLoS ONE (2017) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - free floating section; mouse; loading ...; fig 2e
In order to describe functional and structural pathology-to-network signatures in the brain after cuprizone treatment, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - free floating section on mouse samples (fig 2e). Neuroimage (2017) ncbi
rabbit polyclonal (CT695)
  • western blot; human; loading ...; fig 3b
In order to suggest that loss of the Wnt signaling pathway may contribute to Alzheimer's disease pathogenesis, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on human samples (fig 3b). J Neurochem (2016) ncbi
rabbit polyclonal (CT695)
  • western blot; mouse; loading ...; fig 3a
In order to show that Egr-1 plays role in BACE-1 activation, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on mouse samples (fig 3a). J Biol Chem (2016) ncbi
rabbit polyclonal (CT695)
  • immunocytochemistry; longfin inshore squid; fig 1
In order to characterize organelles that bind microtubules in an ATP dependent manner by studying the amyloid precursor protein of Alzheimer's disease clusters at the organelle/microtubule interface, Invitrogen APP antibody (Invitrogen, 51-C2700) was used in immunocytochemistry on longfin inshore squid samples (fig 1). PLoS ONE (2016) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; mouse; fig 3
In order to assess the deletion of Ccr2 and cavity size and tau pathology after mild traumatic brain injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on mouse samples (fig 3). J Neuroinflammation (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; mouse; 0.75 ug/ul; fig 2
In order to assess Losartan treatment on experimental glaucoma, Invitrogen APP antibody (Novex by Life Technologies, 512700) was used in immunohistochemistry on mouse samples at 0.75 ug/ul (fig 2). PLoS ONE (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; pig; 1:700; fig 6
In order to determine the relationship between injured axons after mild traumatic brain injury in pigs and microglia processes, Invitrogen APP antibody (Life Technologies, 51-2700) was used in immunohistochemistry on pig samples at 1:700 (fig 6). J Neuroinflammation (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; fig 3
In order to elucidate the mechanisms by which increased LMNB1 levels cause autosomal dominant leukodystrophy, Invitrogen APP antibody (Invitrogen, 512700) was used in immunohistochemistry - frozen section on mouse samples (fig 3). J Neurosci (2015) ncbi
rabbit polyclonal (CT695)
  • western blot; human
In order to show that IDN5706 promotes iAPP degradation by activating Atg5-dependent autophagy, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on human samples . PLoS ONE (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; mouse
In order to describe the development of a closed-body model of spinal cord injury in mice, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on mouse samples . Exp Neurol (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; 1:1000; fig 6
  • immunohistochemistry; mouse; 1:50; fig 5
In order to discuss the limitations assessing axonal injury by microscopy, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - frozen section on mouse samples at 1:1000 (fig 6) and in immunohistochemistry on mouse samples at 1:50 (fig 5). J Neurosci Methods (2015) ncbi
rabbit polyclonal (CT695)
  • immunocytochemistry; mouse; 1:100; fig 2
In order to investigate the contribution of TREM2 to microglia function in vivo, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunocytochemistry on mouse samples at 1:100 (fig 2). Acta Neuropathol (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - free floating section; mouse; 1:700
In order to characterize axonal injury over time, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - free floating section on mouse samples at 1:700. Acta Neuropathol (2015) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; rat; 1:400
In order to examine the role of the monoaminergic system in mild blast-induced traumatic brain injury, Invitrogen APP antibody (Life Technologies, 51-2700) was used in immunohistochemistry - frozen section on rat samples at 1:400. J Neurotrauma (2015) ncbi
rabbit polyclonal (CT695)
  • western blot; mouse; 1:500
In order to identify the relation between fragile X mental retardation protein and Alzheimer disease, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on mouse samples at 1:500. Front Genet (2014) ncbi
rabbit polyclonal (CT695)
  • western blot; rat; fig 1
In order to report that Kal9 and Kal12 mediate dendritic maturation in early development, Invitrogen APP antibody (Life Technologies, 51-2700) was used in western blot on rat samples (fig 1). Cereb Cortex (2015) ncbi
rabbit polyclonal (CT695)
  • western blot; human
In order to discuss the effects of BMS-869780, a gamma secretase inhibitor, in several animal models, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on human samples . Int J Alzheimers Dis (2014) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - free floating section; rat; 1:1000
In order to assess if subischemic levels of elevated intracranial pressure without hematoma/contusion contribute to increased morbidity/mortality, Invitrogen APP antibody (Life Technologies, 51-2700) was used in immunohistochemistry - free floating section on rat samples at 1:1000. J Cereb Blood Flow Metab (2014) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; 1:1000
In order to study the contribution of microglia to axon degeneration after repetitive closed-skull traumatic brain injury using CD11b-thymidine kinase mice, Invitrogen APP antibody (Invitrogen, 512700) was used in immunohistochemistry - frozen section on mouse samples at 1:1000. J Neurotrauma (2014) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; 1:2000; fig 6
In order to assess temporal and spatial onset of the reactive glia response in a clinically relevant form of Pelizaeus-Merzbacher disease, Invitrogen APP antibody (Life Technologies, 51-2700) was used in immunohistochemistry - frozen section on mouse samples at 1:2000 (fig 6). J Neuroinflammation (2013) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; 1:1000
In order to study Abeta agggregation and oligomerization induced in a murine model of Alzheimer's disease by experimental traumatic brain injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - frozen section on mouse samples at 1:1000. J Neurotrauma (2014) ncbi
rabbit polyclonal (CT695)
  • western blot; rat; fig 3
In order to assess myelin loss and oligodendrocyte death following traumatic brain injury, Invitrogen APP antibody (Invitrogen, 51 2700) was used in western blot on rat samples (fig 3). Eur J Neurosci (2013) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1:1000; fig 2
In order to study the neuronal somatic and axonal damage that occurs during elevated intracranial pressure, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on rat samples at 1:1000 (fig 2). J Cereb Blood Flow Metab (2012) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; fig 4
In order to investigate diffuse axonal injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on rat samples (fig 4). J Trauma Acute Care Surg (2012) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1 ug/ml; fig 6
In order to perform a time course to study impaired axoplasmic transport and neurofilament compaction, Invitrogen APP antibody (Zymed, 51-2700) was used in immunohistochemistry on rat samples at 1 ug/ml (fig 6). Brain Res (2012) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1:200; fig 4
In order to discuss the use of helmets to prevent traumatic brain injury, Invitrogen APP antibody (Zymed, 51-2700) was used in immunohistochemistry on rat samples at 1:200 (fig 4). Neurosurgery (2012) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1 ug/ml; fig 7, 8
In order to study traumatic axonal injury in rats, Invitrogen APP antibody (Zymed, 51-2700) was used in immunohistochemistry on rat samples at 1 ug/ml (fig 7, 8). J Neurotrauma (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1:100
In order to characterize a new rat model of diffuse brain injury in the sagittal plane, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on rat samples at 1:100. Front Neurol (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; rat; 1:200; fig 7
In order to characterize a rodent model of direct cranial blast injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - frozen section on rat samples at 1:200 (fig 7). J Neurotrauma (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - frozen section; mouse; 1:200; fig 3
In order to investigate the effects of blast injury on the parenchymatous organs of mice, Invitrogen APP antibody (Zymed , 51-2700) was used in immunohistochemistry - frozen section on mouse samples at 1:200 (fig 3). J Neuropathol Exp Neurol (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; human; 1:100; fig 5
In order to examine the oligodendrogenic potential of different human fetal forebrain regions during the first trimester of gestation, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on human samples at 1:100 (fig 5). Brain (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry - free floating section; mouse; 1:100
In order to investigate the effects of using either a rounded- or a flat-tip impactor to apply controlled cortical impact injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry - free floating section on mouse samples at 1:100. J Neurotrauma (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1:200; fig 1
In order to assess the benefits of O3FA supplementation following diffuse axonal injury in rats, Invitrogen APP antibody (Zymed, 51-2700) was used in immunohistochemistry on rat samples at 1:200 (fig 1). J Neurosurg (2011) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; rat; 1:200; fig 2
In order to assess the benefits of DHA treatment following diffuse axonal injury in rats, Invitrogen APP antibody (Zymed, 51-2700) was used in immunohistochemistry on rat samples at 1:200 (fig 2). J Neurotrauma (2010) ncbi
rabbit polyclonal (CT695)
  • western blot; mouse; 1:1000; fig 5
In order to elucidate the mechanism of calpain during Alzheimer's disease progression, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on mouse samples at 1:1000 (fig 5). J Biol Chem (2010) ncbi
rabbit polyclonal (CT695)
  • western blot; mouse; 0.5 ug/ml
In order to test if proprotein convertase subtilisin/kexin type 9 modulates the levels of low-density lipoprotein receptor and other membrane-bound proteins in the adult mouse brain, Invitrogen APP antibody (Invitrogen, 51-2700) was used in western blot on mouse samples at 0.5 ug/ml. J Lipid Res (2010) ncbi
rabbit polyclonal (CT695)
  • immunohistochemistry; mouse; 1:1000; fig 1
In order to assess the effects of acute treatment with minocycline on traumatic brain injury, Invitrogen APP antibody (Invitrogen, 51-2700) was used in immunohistochemistry on mouse samples at 1:1000 (fig 1). J Neurotrauma (2010) ncbi
rabbit polyclonal (CT695)
In order to investigate the regulatory link between beta-amyloid precursor protein and dual-specificity tyrosine(Y)-phosphorylation regulated kinase 1A in Down syndrome brains, Invitrogen APP antibody (Zymed Laboratories, 51-2700) was used . J Neurochem (2008) ncbi
rabbit polyclonal (CT695)
In order to discuss features present in feline myopathy, Invitrogen APP antibody (Zymed, 51?C2700) was used . Acta Neuropathol (2007) ncbi
rabbit polyclonal (CT695)
In order to examine the interaction between amyloid precursor protein and ShcA/Grb2, Invitrogen APP antibody (Zymed Laboratories, 51-2700) was used . Ann N Y Acad Sci (2006) ncbi
rabbit polyclonal (CT695)
In order to propose that amyloid precursor protein and presenilin1 are part of a common signaling pathway that regulates ERK1,2 and the cell cycle, Invitrogen APP antibody (Zymed, 51-2700) was used . J Biol Chem (2007) ncbi
rabbit polyclonal (CT695)
In order to elucidate the molecular mechanism that links Alzheimer disease and fragile X syndrome, Invitrogen APP antibody (Zymed, 51-2700) was used . PLoS Biol (2007) ncbi
rabbit polyclonal (CT695)
In order to develop and characterize a tissue culture model of traumatic brain injury, Invitrogen APP antibody (Zymed, 51-2700) was used . Stapp Car Crash J (2003) ncbi
rabbit polyclonal (CT695)
In order to develop a new weight-drop head-injury apparatus and assess the treated rats, Invitrogen APP antibody (Zymed, 51-2700) was used . J Neurosci Methods (2006) ncbi
Articles Reviewed
  1. Kallakuri S, Desai A, Feng K, Tummala S, Saif T, Chen C, et al. Neuronal Injury and Glial Changes Are Hallmarks of Open Field Blast Exposure in Swine Frontal Lobe. PLoS ONE. 2017;12:e0169239 pubmed publisher
  2. Hübner N, Mechling A, Lee H, Reisert M, Bienert T, Hennig J, et al. The connectomics of brain demyelination: Functional and structural patterns in the cuprizone mouse model. Neuroimage. 2017;146:1-18 pubmed publisher
  3. Tapia Rojas C, Burgos P, Inestrosa N. Inhibition of Wnt signaling induces amyloidogenic processing of amyloid precursor protein and the production and aggregation of Amyloid-? (A?)42 peptides. J Neurochem. 2016;139:1175-1191 pubmed publisher
  4. Qin X, Wang Y, Paudel H. Early Growth Response 1 (Egr-1) Is a Transcriptional Activator of ?-Secretase 1 (BACE-1) in the Brain. J Biol Chem. 2016;291:22276-22287 pubmed
  5. Stevenson J, Conaty E, Walsh R, Poidomani P, Samoriski C, Scollins B, et al. The Amyloid Precursor Protein of Alzheimer's Disease Clusters at the Organelle/Microtubule Interface on Organelles that Bind Microtubules in an ATP Dependent Manner. PLoS ONE. 2016;11:e0147808 pubmed publisher
  6. Gyoneva S, Kim D, Katsumoto A, Kokiko Cochran O, Lamb B, Ransohoff R. Ccr2 deletion dissociates cavity size and tau pathology after mild traumatic brain injury. J Neuroinflammation. 2015;12:228 pubmed publisher
  7. Quigley H, Pitha I, Welsbie D, Nguyen C, Steinhart M, Nguyen T, et al. Losartan Treatment Protects Retinal Ganglion Cells and Alters Scleral Remodeling in Experimental Glaucoma. PLoS ONE. 2015;10:e0141137 pubmed publisher
  8. Lafrenaye A, Todani M, Walker S, Povlishock J. Microglia processes associate with diffusely injured axons following mild traumatic brain injury in the micro pig. J Neuroinflammation. 2015;12:186 pubmed publisher
  9. Rolyan H, Tyurina Y, Hernandez M, Amoscato A, Sparvero L, Nmezi B, et al. Defects of Lipid Synthesis Are Linked to the Age-Dependent Demyelination Caused by Lamin B1 Overexpression. J Neurosci. 2015;35:12002-17 pubmed publisher
  10. Cavieres V, González A, Muñoz V, Yefi C, Bustamante H, Barraza R, et al. Tetrahydrohyperforin Inhibits the Proteolytic Processing of Amyloid Precursor Protein and Enhances Its Degradation by Atg5-Dependent Autophagy. PLoS ONE. 2015;10:e0136313 pubmed publisher
  11. Del Mar N, von Buttlar X, Yu A, Guley N, Reiner A, Honig M. A novel closed-body model of spinal cord injury caused by high-pressure air blasts produces extensive axonal injury and motor impairments. Exp Neurol. 2015;271:53-71 pubmed publisher
  12. Bennett R, Brody D. Array tomography for the detection of non-dilated, injured axons in traumatic brain injury. J Neurosci Methods. 2015;245:25-36 pubmed publisher
  13. Cantoni C, Bollman B, Licastro D, Xie M, Mikesell R, Schmidt R, et al. TREM2 regulates microglial cell activation in response to demyelination in vivo. Acta Neuropathol. 2015;129:429-47 pubmed publisher
  14. HÃ¥nell A, Greer J, McGinn M, Povlishock J. Traumatic brain injury-induced axonal phenotypes react differently to treatment. Acta Neuropathol. 2015;129:317-32 pubmed publisher
  15. Kawa L, Arborelius U, Yoshitake T, Kehr J, Hökfelt T, Risling M, et al. Neurotransmitter Systems in a Mild Blast Traumatic Brain Injury Model: Catecholamines and Serotonin. J Neurotrauma. 2015;32:1190-9 pubmed publisher
  16. Renoux A, Carducci N, Ahmady A, Todd P. Fragile X mental retardation protein expression in Alzheimer's disease. Front Genet. 2014;5:360 pubmed publisher
  17. Yan Y, Eipper B, Mains R. Kalirin-9 and Kalirin-12 Play Essential Roles in Dendritic Outgrowth and Branching. Cereb Cortex. 2015;25:3487-501 pubmed publisher
  18. Toyn J, Thompson L, Lentz K, Meredith J, Burton C, Sankaranararyanan S, et al. Identification and Preclinical Pharmacology of the ?-Secretase Modulator BMS-869780. Int J Alzheimers Dis. 2014;2014:431858 pubmed publisher
  19. Lafrenaye A, Krahe T, Povlishock J. Moderately elevated intracranial pressure after diffuse traumatic brain injury is associated with exacerbated neuronal pathology and behavioral morbidity in the rat. J Cereb Blood Flow Metab. 2014;34:1628-36 pubmed publisher
  20. Bennett R, Brody D. Acute reduction of microglia does not alter axonal injury in a mouse model of repetitive concussive traumatic brain injury. J Neurotrauma. 2014;31:1647-63 pubmed publisher
  21. Bachstetter A, Webster S, Van Eldik L, Cambi F. Clinically relevant intronic splicing enhancer mutation in myelin proteolipid protein leads to progressive microglia and astrocyte activation in white and gray matter regions of the brain. J Neuroinflammation. 2013;10:146 pubmed publisher
  22. Washington P, Morffy N, Parsadanian M, Zapple D, Burns M. Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model. J Neurotrauma. 2014;31:125-34 pubmed publisher
  23. Flygt J, Djupsjö A, Lenne F, Marklund N. Myelin loss and oligodendrocyte pathology in white matter tracts following traumatic brain injury in the rat. Eur J Neurosci. 2013;38:2153-65 pubmed publisher
  24. Lafrenaye A, McGinn M, Povlishock J. Increased intracranial pressure after diffuse traumatic brain injury exacerbates neuronal somatic membrane poration but not axonal injury: evidence for primary intracranial pressure-induced neuronal perturbation. J Cereb Blood Flow Metab. 2012;32:1919-32 pubmed publisher
  25. Jia X, Cong B, Wang S, Dong L, Ma C, Li Y. Secondary damage caused by CD11b+ microglia following diffuse axonal injury in rats. J Trauma Acute Care Surg. 2012;73:1168-74 pubmed
  26. Zakaria N, Kallakuri S, Bandaru S, Cavanaugh J. Temporal assessment of traumatic axonal injury in the rat corpus callosum and optic chiasm. Brain Res. 2012;1467:81-90 pubmed publisher
  27. Smith D, Bailes J, Fisher J, Robles J, Turner R, Mills J. Internal jugular vein compression mitigates traumatic axonal injury in a rat model by reducing the intracranial slosh effect. Neurosurgery. 2012;70:740-6 pubmed publisher
  28. Li Y, Zhang L, Kallakuri S, Zhou R, Cavanaugh J. Quantitative relationship between axonal injury and mechanical response in a rodent head impact acceleration model. J Neurotrauma. 2011;28:1767-82 pubmed publisher
  29. Davidsson J, Risling M. A new model to produce sagittal plane rotational induced diffuse axonal injuries. Front Neurol. 2011;2:41 pubmed publisher
  30. Kuehn R, Simard P, Driscoll I, Keledjian K, Ivanova S, Tosun C, et al. Rodent model of direct cranial blast injury. J Neurotrauma. 2011;28:2155-69 pubmed publisher
  31. Koliatsos V, Cernak I, Xu L, Song Y, Savonenko A, Crain B, et al. A mouse model of blast injury to brain: initial pathological, neuropathological, and behavioral characterization. J Neuropathol Exp Neurol. 2011;70:399-416 pubmed publisher
  32. Buchet D, Garcia C, Deboux C, Nait Oumesmar B, Baron van Evercooren A. Human neural progenitors from different foetal forebrain regions remyelinate the adult mouse spinal cord. Brain. 2011;134:1168-83 pubmed publisher
  33. Pleasant J, Carlson S, Mao H, Scheff S, Yang K, Saatman K. Rate of neurodegeneration in the mouse controlled cortical impact model is influenced by impactor tip shape: implications for mechanistic and therapeutic studies. J Neurotrauma. 2011;28:2245-62 pubmed publisher
  34. Mills J, Bailes J, Sedney C, Hutchins H, Sears B. Omega-3 fatty acid supplementation and reduction of traumatic axonal injury in a rodent head injury model. J Neurosurg. 2011;114:77-84 pubmed publisher
  35. Bailes J, Mills J. Docosahexaenoic acid reduces traumatic axonal injury in a rodent head injury model. J Neurotrauma. 2010;27:1617-24 pubmed publisher
  36. Liang B, Duan B, Zhou X, Gong J, Luo Z. Calpain activation promotes BACE1 expression, amyloid precursor protein processing, and amyloid plaque formation in a transgenic mouse model of Alzheimer disease. J Biol Chem. 2010;285:27737-44 pubmed publisher
  37. Liu M, Wu G, Baysarowich J, Kavana M, Addona G, Bierilo K, et al. PCSK9 is not involved in the degradation of LDL receptors and BACE1 in the adult mouse brain. J Lipid Res. 2010;51:2611-8 pubmed publisher
  38. Homsi S, Piaggio T, Croci N, Noble F, Plotkine M, Marchand Leroux C, et al. Blockade of acute microglial activation by minocycline promotes neuroprotection and reduces locomotor hyperactivity after closed head injury in mice: a twelve-week follow-up study. J Neurotrauma. 2010;27:911-21 pubmed publisher
  39. Ryoo S, Cho H, Lee H, Jeong H, Radnaabazar C, Kim Y, et al. Dual-specificity tyrosine(Y)-phosphorylation regulated kinase 1A-mediated phosphorylation of amyloid precursor protein: evidence for a functional link between Down syndrome and Alzheimer's disease. J Neurochem. 2008;104:1333-44 pubmed
  40. Shelton G, Sturges B, Lyons L, Williams D, Aleman M, Jiang Y, et al. Myopathy with tubulin-reactive inclusions in two cats. Acta Neuropathol. 2007;114:537-42 pubmed
  41. Venezia V, Nizzari M, Repetto E, Violani E, Corsaro A, Thellung S, et al. Amyloid precursor protein modulates ERK-1 and -2 signaling. Ann N Y Acad Sci. 2006;1090:455-65 pubmed
  42. Nizzari M, Venezia V, Repetto E, Caorsi V, Magrassi R, Gagliani M, et al. Amyloid precursor protein and Presenilin1 interact with the adaptor GRB2 and modulate ERK 1,2 signaling. J Biol Chem. 2007;282:13833-44 pubmed
  43. Westmark C, Malter J. FMRP mediates mGluR5-dependent translation of amyloid precursor protein. PLoS Biol. 2007;5:e52 pubmed
  44. Morrison B, Cater H, Wang C, Thomas F, Hung C, Ateshian G, et al. A tissue level tolerance criterion for living brain developed with an in vitro model of traumatic mechanical loading. Stapp Car Crash J. 2003;47:93-105 pubmed
  45. Pal J, Toth Z, Farkas O, Kellenyi L, Doczi T, Gallyas F. Selective induction of ultrastructural (neurofilament) compaction in axons by means of a new head-injury apparatus. J Neurosci Methods. 2006;153:283-9 pubmed