This is a Validated Antibody Database (VAD) review about human SNTB2, based on 3 published articles (read how Labome selects the articles), using SNTB2 antibody in all methods. It is aimed to help Labome visitors find the most suited SNTB2 antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
SNTB2 synonym: D16S2531E; EST25263; SNT2B2; SNT3; SNTL; beta-2-syntrophin; 59 kDa dystrophin-associated protein A1 basic component 2; dystrophin-associated protein A1, 59kD, basic component 2; syntrophin, beta 2 (dystrophin-associated protein A1, 59kDa, basic component 2); syntrophin-3

Invitrogen
mouse monoclonal (1351)
  • western blot; human; 1:200; fig 2
In order to investigate the role of Kir4.1 variants to autism-epilepsy comorbidity, Invitrogen SNTB2 antibody (Affinity BioReagents, MA-1-745) was used in western blot on human samples at 1:200 (fig 2). Sci Rep (2016) ncbi
mouse monoclonal (1351)
  • immunohistochemistry - frozen section; mouse; 1:100; fig 3
In order to study the localization of neuronal nitric oxide synthase to utrophin expressing intercalated discs and stabilization of their structural integrity, Invitrogen SNTB2 antibody (Pierce, MA1-745) was used in immunohistochemistry - frozen section on mouse samples at 1:100 (fig 3). Neuromuscul Disord (2015) ncbi
mouse monoclonal (1351)
  • western blot; mouse; fig 7
In order to investigate the role of glial alpha-dystrobrevin in the breakdown of the blood-brain barrier and progressive brain edema, Invitrogen SNTB2 antibody (Affinity BioReagents, 1351) was used in western blot on mouse samples (fig 7). J Biol Chem (2012) ncbi
Articles Reviewed
  1. Sicca F, Ambrosini E, Marchese M, Sforna L, Servettini I, Valvo G, et al. Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy. Sci Rep. 2016;6:34325 pubmed publisher
  2. Gonzalez J, Crassous P, Schneider J, Beuve A, Fraidenraich D. Neuronal nitric oxide synthase localizes to utrophin expressing intercalated discs and stabilizes their structural integrity. Neuromuscul Disord. 2015;25:964-76 pubmed publisher
  3. Lien C, Mohanta S, Frontczak Baniewicz M, Swinny J, Zablocka B, Gorecki D. Absence of glial ?-dystrobrevin causes abnormalities of the blood-brain barrier and progressive brain edema. J Biol Chem. 2012;287:41374-85 pubmed publisher