This is a Validated Antibody Database (VAD) review about human MIEF2, based on 3 published articles (read how Labome selects the articles), using MIEF2 antibody in all methods. It is aimed to help Labome visitors find the most suited MIEF2 antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
MIEF2 synonym: MID49; SMCR7

Knockout validation
Proteintech Group
domestic rabbit polyclonal
  • western blot knockout validation; human; fig 1a
Proteintech Group MIEF2 antibody (Proteintech, noca) was used in western blot knockout validation on human samples (fig 1a). J Cell Biol (2016) ncbi
Proteintech Group
domestic rabbit polyclonal
  • western blot knockout validation; human; fig 1a
Proteintech Group MIEF2 antibody (Proteintech, noca) was used in western blot knockout validation on human samples (fig 1a). J Cell Biol (2016) ncbi
domestic rabbit polyclonal
  • western blot; mouse; fig s2
In order to analyze delay of neurodegeneration by preventing stress-induced OPA1 processing in mitochondria by loss of OMA1, Proteintech Group MIEF2 antibody (ProteinTech, 16413-1-AP) was used in western blot on mouse samples (fig s2). J Cell Biol (2016) ncbi
Abcam
domestic rabbit polyclonal
  • western blot; mouse; 1:1000; loading ...; fig 5e
Abcam MIEF2 antibody (Abcam, ab101350) was used in western blot on mouse samples at 1:1000 (fig 5e). Redox Biol (2019) ncbi
Articles Reviewed
  1. Li H, Feng J, Zhang Y, Feng J, Wang Q, Zhao S, et al. Mst1 deletion attenuates renal ischaemia-reperfusion injury: The role of microtubule cytoskeleton dynamics, mitochondrial fission and the GSK3β-p53 signalling pathway. Redox Biol. 2019;20:261-274 pubmed publisher
  2. Otera H, Miyata N, Kuge O, Mihara K. Drp1-dependent mitochondrial fission via MiD49/51 is essential for apoptotic cristae remodeling. J Cell Biol. 2016;212:531-44 pubmed publisher
  3. Korwitz A, Merkwirth C, Richter Dennerlein R, Tröder S, Sprenger H, Quirós P, et al. Loss of OMA1 delays neurodegeneration by preventing stress-induced OPA1 processing in mitochondria. J Cell Biol. 2016;212:157-66 pubmed publisher