This is a Validated Antibody Database (VAD) review about human H3-5, based on 14 published articles (read how Labome selects the articles), using H3-5 antibody in all methods. It is aimed to help Labome visitors find the most suited H3-5 antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
H3-5 synonym: H3.5; H3F3C

Invitrogen
domestic rabbit recombinant (24H8L19)
  • western blot; human; 1:1000; fig 2a
Invitrogen H3-5 antibody (Thermo-fisher scientific, 701764) was used in western blot on human samples at 1:1000 (fig 2a). Ann Med (2021) ncbi
domestic rabbit polyclonal
  • ChIP-Seq; human; loading ...; fig 5b
Invitrogen H3-5 antibody (Life Technologies, 49-1010) was used in ChIP-Seq on human samples (fig 5b). Cancer Cell (2018) ncbi
domestic rabbit polyclonal
  • ChIP-Seq; human; loading ...; fig 5b
Invitrogen H3-5 antibody (Invitrogen, 491008) was used in ChIP-Seq on human samples (fig 5b). Cancer Cell (2018) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; loading ...; fig 6c
In order to test if posterior HOXD gene activation and Ewing sarcoma tumorigenicity are both regulated by MLL1 and/or menin, Invitrogen H3-5 antibody (Invitrogen, 49-1005) was used in chromatin immunoprecipitation on human samples (fig 6c). Oncotarget (2017) ncbi
domestic rabbit polyclonal
  • ChIP-Seq; human; fig 1
In order to investigate the evolutionary origin of decidual stromal cells, Invitrogen H3-5 antibody (Invitrogen, 49-1005) was used in ChIP-Seq on human samples (fig 1). Mol Biol Evol (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; loading ...; fig 3c
In order to test if CXCR4 impacts tumor growth, Invitrogen H3-5 antibody (Life Technologies, 49-1005) was used in chromatin immunoprecipitation on human samples (fig 3c). Oncotarget (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; loading ...; fig 3a
In order to clarify the link between miR-152 and CDH1 function, Invitrogen H3-5 antibody (Invitrogen, 49-1005) was used in chromatin immunoprecipitation on human samples (fig 3a). Exp Cell Res (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; loading ...; fig 3a
In order to clarify the link between miR-152 and CDH1 function, Invitrogen H3-5 antibody (Invitrogen, 49-1003) was used in chromatin immunoprecipitation on human samples (fig 3a). Exp Cell Res (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; loading ...; fig 3a
In order to clarify the link between miR-152 and CDH1 function, Invitrogen H3-5 antibody (Invitrogen, 49-1008) was used in chromatin immunoprecipitation on human samples (fig 3a). Exp Cell Res (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; fig 5
In order to analyze epigenetic drift towards histone modifications and how they regulate CAV1 gene expression in colon cancer, Invitrogen H3-5 antibody (Invitrogen, 49-1005) was used in chromatin immunoprecipitation on human samples (fig 5). Gene (2016) ncbi
domestic rabbit polyclonal
  • chromatin immunoprecipitation; human; fig 5
In order to analyze epigenetic drift towards histone modifications and how they regulate CAV1 gene expression in colon cancer, Invitrogen H3-5 antibody (Invitrogen, 49-1008) was used in chromatin immunoprecipitation on human samples (fig 5). Gene (2016) ncbi
domestic rabbit polyclonal
  • western blot; human; fig 5
In order to study reversal of chemotherapy drug resistance in cervical cancer cells by interference with endogenous EZH2 and up-regulation of Dicer expression, Invitrogen H3-5 antibody (Thermo Scientific, A15024) was used in western blot on human samples (fig 5). Tumour Biol (2016) ncbi
domestic rabbit monoclonal (J.924.2)
  • immunocytochemistry; American tobacco; 1:200; fig 2
In order to study how chromosomal changes contribute to cytomixis, Invitrogen H3-5 antibody (Thermo Scientific, MA5-11195) was used in immunocytochemistry on American tobacco samples at 1:200 (fig 2). Front Plant Sci (2015) ncbi
domestic rabbit polyclonal
In order to develop a novel, programmable transcription factor prototype, Invitrogen H3-5 antibody (Invitrogen, P7N49-1008) was used . Nucleic Acids Res (2015) ncbi
domestic rabbit monoclonal (E.960.2)
  • western blot; human; fig 6
In order to test if celastrol inhibits formation of neutrophil extracellular traps induced by inflammatory stimuli associated with rheumatoid arthritis and systemic lupus erythematosus, Invitrogen H3-5 antibody (Thermo Fisher Scientific, MA5-15150) was used in western blot on human samples (fig 6). Curr Mol Med (2015) ncbi
domestic rabbit monoclonal (G.532.8)
  • chromatin immunoprecipitation; human
In order to study how the cellular changes induced by HSP90 inhibition affect cancer, Invitrogen H3-5 antibody (Thermo, MA511199) was used in chromatin immunoprecipitation on human samples . J Biol Chem (2014) ncbi
Active Motif
rat monoclonal (8H6-2111)
  • immunocytochemistry; human; 1:200; loading ...; fig 4c, 4g, s3d
Active Motif H3-5 antibody (Active Motif, 61161) was used in immunocytochemistry on human samples at 1:200 (fig 4c, 4g, s3d). Cell Stem Cell (2022) ncbi
rat monoclonal (8H6-2111)
  • immunocytochemistry; human; loading ...; fig 3b
  • western blot; human; loading ...; fig 3c
Active Motif H3-5 antibody (Active Motif, 61161) was used in immunocytochemistry on human samples (fig 3b) and in western blot on human samples (fig 3c). Cell Rep (2019) ncbi
Articles Reviewed
  1. Taubenschmid Stowers J, Rostovskaya M, Santos F, Ljung S, Argelaguet R, Krueger F, et al. 8C-like cells capture the human zygotic genome activation program in vitro. Cell Stem Cell. 2022;29:449-459.e6 pubmed publisher
  2. Al Abdulsalam E, Al Harithy R. Visfatin and global histone H3K9me levels in colon cancer. Ann Med. 2021;53:647-652 pubmed publisher
  3. Resnick R, Wong C, Hamm D, Bennett S, Skene P, Hake S, et al. DUX4-Induced Histone Variants H3.X and H3.Y Mark DUX4 Target Genes for Expression. Cell Rep. 2019;29:1812-1820.e5 pubmed publisher
  4. Stewart E, McEvoy J, Wang H, Chen X, Honnell V, Ocarz M, et al. Identification of Therapeutic Targets in Rhabdomyosarcoma through Integrated Genomic, Epigenomic, and Proteomic Analyses. Cancer Cell. 2018;34:411-426.e19 pubmed publisher
  5. Svoboda L, Bailey N, Van Noord R, Krook M, Harris A, Cramer C, et al. Tumorigenicity of Ewing sarcoma is critically dependent on the trithorax proteins MLL1 and menin. Oncotarget. 2017;8:458-471 pubmed publisher
  6. Park Y, Nnamani M, Maziarz J, Wagner G. Cis-Regulatory Evolution of Forkhead Box O1 (FOXO1), a Terminal Selector Gene for Decidual Stromal Cell Identity. Mol Biol Evol. 2016;33:3161-3169 pubmed
  7. Krook M, Hawkins A, Patel R, Lucas D, Van Noord R, Chugh R, et al. A bivalent promoter contributes to stress-induced plasticity of CXCR4 in Ewing sarcoma. Oncotarget. 2016;7:61775-61788 pubmed publisher
  8. Sengupta D, Deb M, Rath S, Kar S, Parbin S, Pradhan N, et al. DNA methylation and not H3K4 trimethylation dictates the expression status of miR-152 gene which inhibits migration of breast cancer cells via DNMT1/CDH1 loop. Exp Cell Res. 2016;346:176-87 pubmed publisher
  9. Deb M, Sengupta D, Kar S, Rath S, Roy S, Das G, et al. Epigenetic drift towards histone modifications regulates CAV1 gene expression in colon cancer. Gene. 2016;581:75-84 pubmed publisher
  10. Cai L, Wang Z, Liu D. Interference with endogenous EZH2 reverses the chemotherapy drug resistance in cervical cancer cells partly by up-regulating Dicer expression. Tumour Biol. 2016;37:6359-69 pubmed publisher
  11. Mursalimov S, Permyakova N, Deineko E, Houben A, Demidov D. Cytomixis doesn't induce obvious changes in chromatin modifications and programmed cell death in tobacco male meiocytes. Front Plant Sci. 2015;6:846 pubmed publisher
  12. Fimiani C, Goina E, Mallamaci A. Upregulating endogenous genes by an RNA-programmable artificial transactivator. Nucleic Acids Res. 2015;43:7850-64 pubmed publisher
  13. Yu Y, Koehn C, Yue Y, Li S, Thiele G, Hearth Holmes M, et al. Celastrol inhibits inflammatory stimuli-induced neutrophil extracellular trap formation. Curr Mol Med. 2015;15:401-10 pubmed
  14. Chen Y, Chen J, Yu J, Yang G, Temple E, Harbinski F, et al. Identification of mixed lineage leukemia 1(MLL1) protein as a coactivator of heat shock factor 1(HSF1) protein in response to heat shock protein 90 (HSP90) inhibition. J Biol Chem. 2014;289:18914-27 pubmed publisher