This is a Validated Antibody Database (VAD) review about cat GJB6, based on 63 published articles (read how Labome selects the articles), using GJB6 antibody in all methods. It is aimed to help Labome visitors find the most suited GJB6 antibody. Please note the number of articles fluctuates since newly identified citations are added and citations for discontinued catalog numbers are removed regularly.
GJB6 synonym: gap junction beta-6 protein; connexin 30

Invitrogen
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:1000; loading ...; fig 3e
Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 1:1000 (fig 3e). J Comp Neurol (2019) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:400; loading ...; fig 1a
Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 1:400 (fig 1a). Int J Mol Sci (2018) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:250; loading ...; fig 1c
In order to assess the mechanism of a gap-junction protein connexin30 mutant to rescue hearing in accelerated age-related hearing loss CD-1 mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 1:250 (fig 1c). Nat Commun (2017) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; loading ...; fig 2l
In order to describe a method to differentiate induced pluripotent stem cells into functional CX26-gap junction plaque-forming cells, Invitrogen GJB6 antibody (Life Technologies, 71-2200) was used in immunohistochemistry on mouse samples (fig 2l). Stem Cell Reports (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:75
  • western blot; mouse; 1:500; fig 3
In order to assess the transiently formed junctional nexuses during post-natal mammary gland development by E-cadherins, connexins, beta-catenin, and claudin-7, Invitrogen GJB6 antibody (Life Technologies, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 1:75 and in western blot on mouse samples at 1:500 (fig 3). Dev Biol (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:100; fig 7
In order to investigate breast tumorigenesis due to loss of Panx1 which impairs mammary gland development at lactation, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 1:100 (fig 7). PLoS ONE (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:200; fig 6
  • western blot; mouse; 1:250; fig 2
In order to study differential expression of astrocytic connexins by a prenatal alcohol exposure mouse model, Invitrogen GJB6 antibody (Invitrogen, 712200) was used in immunohistochemistry - frozen section on mouse samples at 1:200 (fig 6) and in western blot on mouse samples at 1:250 (fig 2). Neurobiol Dis (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; human; 1:100; fig 1
In order to assess the lateral wall of the cochlea and connexin26/30 proteins and separate expression in man by using super-resolution structured illumination fluorescence microscopy, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on human samples at 1:100 (fig 1). Cell Tissue Res (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; human; fig 3
In order to learn the connection between chronic rhinosinusitis and connexin gap junction channels, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on human samples (fig 3). Int Forum Allergy Rhinol (2016) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:400; fig 1
In order to report that miRNAs pass through gap junctions between native cochlear supporting cells to regulate cochlear development, Invitrogen GJB6 antibody (Invitrogen, 71 - 2200) was used in immunohistochemistry on mouse samples at 1:400 (fig 1). Sci Rep (2015) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; human; 1:125
In order to study protein kinase c-mediated regulation of connexin 30 hemichannels, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in western blot on human samples at 1:125. J Neurophysiol (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; human
  • immunocytochemistry; rat
  • western blot; rat
In order to study the synthesis and localization of connexin 30, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunocytochemistry on human samples , in immunocytochemistry on rat samples and in western blot on rat samples . J Cell Sci (2015) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; mouse; 1:500; fig 3c
In order to examine the effects of sleep and wake on astrocytes, Invitrogen GJB6 antibody (Life Technologies, 712200) was used in western blot on mouse samples at 1:500 (fig 3c). BMC Biol (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:200
In order to study vestibular dysfunction and organ pathology in connexin-26 knock out mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 1:200. Hear Res (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:400; fig s2.c
In order to study how Panx1 channels regulate ATP release in the cochlea, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 1:400 (fig s2.c). Sci Rep (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse
In order to investigate the effects of transplanted neural precursor cells in the chronic MOG-induced experimental autoimmune encephalomyelitis model, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples . Glia (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; human; 1:250
  • immunocytochemistry; mouse; 1:250
In order to examine the expression and roles Cx30, Cx36, and Cx43 in neuronal differentiation, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunocytochemistry on human samples at 1:250 and in immunocytochemistry on mouse samples at 1:250. Cell Biol Int Rep (2010) (2013) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; rat
In order to study hearing loss using a time-controlled, inducible gene knockout technique to knockout Cx26 expression in the cochlea, Invitrogen GJB6 antibody (Invitrogen Corp, 71-2200) was used in immunohistochemistry on rat samples . Neuroscience (2015) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; chicken; 1:200
In order to study the role of gap junctional coupling in epithelial repair in the avian cochlea, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunocytochemistry on chicken samples at 1:200. J Neurosci (2014) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; mouse; fig 2
In order to determine if patients with loss-of-function Cx26 mutations have a greater susceptibility to impaired breast development, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in western blot on mouse samples (fig 2). PLoS ONE (2014) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse
  • western blot; mouse
In order to report the consequences of the CLN3 mutation on astrocyte function, Invitrogen GJB6 antibody (Life Technologies, 71-2200) was used in immunohistochemistry - frozen section on mouse samples and in western blot on mouse samples . PLoS ONE (2014) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:400
In order to study the mechanism of deafness induced by Connexin 26 deficiency, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 1:400. Biochem Biophys Res Commun (2014) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:250
In order to investigate the effect of the Clouston syndrome mutation connexin30 A88V on the proliferation of sebaceous glands and hearing in mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 1:250. FEBS Lett (2014) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:500; fig 3i
In order to show that connexin 30 controls hippocampal excitatory synaptic transmission through modulation of astroglial glutamate transport, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunohistochemistry on mouse samples at 1:500 (fig 3i). Nat Neurosci (2014) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; human; 1:500
In order to investigate the expression of gap junction proteins in Dupuytren disease, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on human samples at 1:500. Acta Orthop (2014) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; mouse; fig 1
In order to study the role of improved intercellular gap junction communication in the mechanism by which tanshinone IIA enhances the bystander effect during HSV-thymidine kinase/ganciclovire gene therapy, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in western blot on mouse samples (fig 1). PLoS ONE (2013) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; mouse; 1:250; fig 4
In order to assess the effect of the +/G60S mutation in connexin 43 following stroke, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in western blot on mouse samples at 1:250 (fig 4). Neuropharmacology (2013) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; fig 1
In order to show that gap junctions between cochlear supporting cells have a critical role in active cochlear amplification in vivo, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples (fig 1). Nat Commun (2013) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 1:200; fig 5
In order to determine connexin changes during check wound healing, Invitrogen GJB6 antibody (Invitrogen, 712200) was used in immunohistochemistry - frozen section on mouse samples at 1:200 (fig 5). Wound Repair Regen (2013) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; fig 1
In order to determine the role of connexin 30 in epithelial repair following injury in the organ of Corti, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples (fig 1). J Cell Sci (2013) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; human; 1:250; fig 2
In order to study Cx30 and Cx43 using inducible expression systems, Invitrogen GJB6 antibody (Invitrogen, 712200) was used in immunocytochemistry on human samples at 1:250 (fig 2). Anal Biochem (2012) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; human; 1:200; fig 5
  • western blot; human; 1:1000; fig 5
In order to assess the role of tight junctions and gap proteins in cervical remodeling, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunohistochemistry - frozen section on human samples at 1:200 (fig 5) and in western blot on human samples at 1:1000 (fig 5). J Transl Med (2012) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; mouse; 2 ug/ml
In order to study localization of connexins in the brain, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on mouse samples at 2 ug/ml. Eur J Neurosci (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; fig 3
In order to study the expression and function of connexin26 and connexin30 in the ears of transgenic mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples (fig 3). PLoS ONE (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; mouse; 1:500; fig 3
In order to study connexin 26 and connexin 32 isoforms in the cochlea, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunocytochemistry on mouse samples at 1:500 (fig 3). Eur J Cell Biol (2011) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; rat; 1:500; tbl 1
In order to investigate the role of connexins in the production of reactive oxygen-nitrogen species and astrocytic gap junctional communication in diabetic rats, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in western blot on rat samples at 1:500 (tbl 1). J Neurosci Res (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; fig 5
In order to examine the effects of inflammation in the central nervous system on astrocyte gap junction communication and electrophysiological properties, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples (fig 5). J Neurosci (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; human; 1:1000; fig 4
In order to determine the connexin composition of intracellular channels present in astrocytes and oligodendrocytes, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunocytochemistry on human samples at 1:1000 (fig 4). Glia (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:500; fig 5
In order to study the Cx26S17F mutation in mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 1:500 (fig 5). Hum Mol Genet (2011) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 2.5 ug/ml; fig 4
  • western blot; mouse; 1:500; fig 3
In order to create and characterize a mouse model for human bilateral middle/high-frequency hearing loss, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry on mouse samples at 2.5 ug/ml (fig 4) and in western blot on mouse samples at 1:500 (fig 3). Hum Mol Genet (2010) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry; mouse; 1:500; fig 3
In order to determine the expression pattern of connexin43 and 30 in two murine models of Alzheimer disease, Invitrogen GJB6 antibody (Zymed, 712200) was used in immunohistochemistry on mouse samples at 1:500 (fig 3). Neuroscience (2010) ncbi
rabbit polyclonal (Z-PP9)
  • western blot; rat; fig 3
In order to test if hippocampal astrocytes have altered function soon after kainic acid-induced status epilepticus, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in western blot on rat samples (fig 3). Neurobiol Dis (2010) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - frozen section; rat; 1-5 ug/ml; fig 8
In order to investigate the influence of experimental diabetes on gap junction channel-mediated dye transfer, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used in immunohistochemistry - frozen section on rat samples at 1-5 ug/ml (fig 8). ASN Neuro (2010) ncbi
rabbit polyclonal (Z-PP9)
  • immunocytochemistry; human; 1:1000; fig 4
In order to test if dominant Cx26 mutants interact directly with Cx30, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunocytochemistry on human samples at 1:1000 (fig 4). Neurobiol Dis (2010) ncbi
rabbit polyclonal (Z-PP9)
  • immunohistochemistry - paraffin section; human; 1:40; fig 2
In order to study Cx26, Cx30, and Cx43 expression in the epidermal microenvironment of melanocytic nevi and melanomas, Invitrogen GJB6 antibody (Zymed, 71-2200) was used in immunohistochemistry - paraffin section on human samples at 1:40 (fig 2). Histochem Cell Biol (2010) ncbi
rabbit polyclonal (Z-PP9)
In order to examine gap junction-mediated ionic and metabolic coupling in the cochlea of connexin-30 knock out mice, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . PLoS ONE (2008) ncbi
rabbit polyclonal (Z-PP9)
In order to study the extent of coupling in microvessels of the rodent brainstem and retina and the subtypes of intracellular calcium stores that mediate astrocytic signaling, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . J Comp Neurol (2008) ncbi
rabbit polyclonal (Z-PP9)
In order to examine the cellular expression of connexin 26 and 30 in the cochlear lateral wall of rats and guinea pigs, Invitrogen GJB6 antibody (Zymed Laboratories, 71-2200) was used . Cell Tissue Res (2008) ncbi
rabbit polyclonal (Z-PP9)
In order to test if astrocyte connexins are regulated by neurons, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Glia (2008) ncbi
rabbit polyclonal (Z-PP9)
In order to test if vestibular hair cells are degraded in connexin knockout mice, Invitrogen GJB6 antibody (Invitrogen, 71-2200) was used . J Comp Neurol (2007) ncbi
rabbit polyclonal (Z-PP9)
In order to investigate the role of Fbx2 in ear homeostasis, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . J Neurosci (2007) ncbi
rabbit polyclonal (Z-PP9)
In order to study Cx26 and Cx30 in the cochlear sensory epithelium of guinea pigs, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . J Comp Neurol (2006) ncbi
rabbit polyclonal (Z-PP9)
In order to study connexins in mammalian Muller cells, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Vis Neurosci (2006) ncbi
rabbit polyclonal (Z-PP9)
In order to investigate the development of astrocytes in the cat visual cortex, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Brain Res Dev Brain Res (2005) ncbi
rabbit polyclonal (Z-PP9)
In order to report that connexin 26 and connexin 30 induced in keratinocyte-derived epithelial skin tumors, Invitrogen GJB6 antibody (Zymed, 71?C2200) was used . J Histochem Cytochem (2006) ncbi
rabbit polyclonal (Z-PP9)
In order to investigate the selectivity of connexin hemichannels and gap junctions in the guinea pig cochlea, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Eur J Neurosci (2005) ncbi
rabbit polyclonal (Z-PP9)
In order to examine the connexin associations at oligodendrocyte/astrocyte gap junctions on oligodendrocytes in normal and Cx32 knockout mice, Invitrogen GJB6 antibody (Zymed, 71?C2200) was used . Glia (2003) ncbi
rabbit polyclonal (Z-PP9)
In order to examine the relationships among HPV infection, gap junctions, and tumor progression, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Oncogene (2003) ncbi
rabbit polyclonal (Z-PP9)
In order to study the role of connexin 26 in dominantly inherited skin disease, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Hum Mol Genet (2003) ncbi
rabbit polyclonal (Z-PP9)
In order to study connexins in retinal glial cells, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . J Comp Neurol (2003) ncbi
rabbit polyclonal (Z-PP9)
In order to study postnatal development of neuronal projections using cats, Invitrogen GJB6 antibody (Zymed, 71?C2200) was used . J Physiol Paris (2002) ncbi
rabbit polyclonal (Z-PP9)
In order to assess the regional, cellular, and subcellular localization of Cx26 in mouse and rat neural tissue, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . J Comp Neurol (2001) ncbi
rabbit polyclonal (Z-PP9)
In order to measure astrocytic gap junction proteins in young and senescent brains and quantify the extent of functional gap junction coupling, Invitrogen GJB6 antibody (Zymed, 71-2200) was used . Brain Res (2001) ncbi
Articles Reviewed
  1. Piantanida A, Acosta L, Brocardo L, Capurro C, Greer C, Rela L. Selective Cre-mediated gene deletion identifies connexin 43 as the main connexin channel supporting olfactory ensheathing cell networks. J Comp Neurol. 2019;527:1278-1289 pubmed publisher
  2. Chen J, Liang C, Zong L, Zhu Y, Zhao H. Knockout of Pannexin-1 Induces Hearing Loss. Int J Mol Sci. 2018;19: pubmed publisher
  3. Lukashkina V, Levic S, Lukashkin A, Strenzke N, Russell I. A connexin30 mutation rescues hearing and reveals roles for gap junctions in cochlear amplification and micromechanics. Nat Commun. 2017;8:14530 pubmed publisher
  4. Fukunaga I, Fujimoto A, Hatakeyama K, Aoki T, Nishikawa A, Noda T, et al. In Vitro Models of GJB2-Related Hearing Loss Recapitulate Ca2+ Transients via a Gap Junction Characteristic of Developing Cochlea. Stem Cell Reports. 2016;7:1023-1036 pubmed publisher
  5. Dianati E, Poiraud J, Weber Ouellette A, Plante I. Connexins, E-cadherin, Claudin-7 and ?-catenin transiently form junctional nexuses during the post-natal mammary gland development. Dev Biol. 2016;416:52-68 pubmed publisher
  6. Stewart M, Plante I, Penuela S, Laird D. Loss of Panx1 Impairs Mammary Gland Development at Lactation: Implications for Breast Tumorigenesis. PLoS ONE. 2016;11:e0154162 pubmed publisher
  7. Ramani M, Mylvaganam S, Krawczyk M, Wang L, Zoidl C, Brien J, et al. Differential expression of astrocytic connexins in a mouse model of prenatal alcohol exposure. Neurobiol Dis. 2016;91:83-93 pubmed publisher
  8. Liu W, Edin F, Blom H, Magnusson P, Schrott Fischer A, Glueckert R, et al. Super-resolution structured illumination fluorescence microscopy of the lateral wall of the cochlea: the Connexin26/30 proteins are separately expressed in man. Cell Tissue Res. 2016;365:13-27 pubmed publisher
  9. Kim R, Chang G, Hu R, Phillips A, Douglas R. Connexin gap junction channels and chronic rhinosinusitis. Int Forum Allergy Rhinol. 2016;6:611-7 pubmed publisher
  10. Zhu Y, Zong L, Mei L, Zhao H. Connexin26 gap junction mediates miRNA intercellular genetic communication in the cochlea and is required for inner ear development. Sci Rep. 2015;5:15647 pubmed publisher
  11. Alstrøm J, Hansen D, Nielsen M, MacAulay N. Isoform-specific phosphorylation-dependent regulation of connexin hemichannels. J Neurophysiol. 2015;114:3014-22 pubmed publisher
  12. Kelly J, Shao Q, Jagger D, Laird D. Cx30 exhibits unique characteristics including a long half-life when assembled into gap junctions. J Cell Sci. 2015;128:3947-60 pubmed publisher
  13. Bellesi M, de Vivo L, Tononi G, Cirelli C. Effects of sleep and wake on astrocytes: clues from molecular and ultrastructural studies. BMC Biol. 2015;13:66 pubmed publisher
  14. Lee M, Takada T, Takada Y, Kappy M, Beyer L, Swiderski D, et al. Mice with conditional deletion of Cx26 exhibit no vestibular phenotype despite secondary loss of Cx30 in the vestibular end organs. Hear Res. 2015;328:102-12 pubmed publisher
  15. Chen J, Zhu Y, Liang C, Chen J, Zhao H. Pannexin1 channels dominate ATP release in the cochlea ensuring endocochlear potential and auditory receptor potential generation and hearing. Sci Rep. 2015;5:10762 pubmed publisher
  16. Theotokis P, Kleopa K, Touloumi O, Lagoudaki R, Lourbopoulos A, Nousiopoulou E, et al. Connexin43 and connexin47 alterations after neural precursor cells transplantation in experimental autoimmune encephalomyelitis. Glia. 2015;63:1772-83 pubmed publisher
  17. Wan C, O Carroll S, Kim S, Green C, Nicholson L. Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells. Cell Biol Int Rep (2010). 2013;20:13-23 pubmed
  18. Zhu Y, Liang C, Zong L, Chen J, Jones R, Zhao H. Connexin26 (GJB2) deficiency reduces active cochlear amplification leading to late-onset hearing loss. Neuroscience. 2015;284:719-29 pubmed publisher
  19. Jagger D, Nickel R, Forge A. Gap junctional coupling is essential for epithelial repair in the avian cochlea. J Neurosci. 2014;34:15851-60 pubmed publisher
  20. Stewart M, Plante I, Bechberger J, Naus C, Laird D. Mammary gland specific knockdown of the physiological surge in Cx26 during lactation retains normal mammary gland development and function. PLoS ONE. 2014;9:e101546 pubmed publisher
  21. Burkovetskaya M, Karpuk N, Xiong J, Bosch M, Boska M, Takeuchi H, et al. Evidence for aberrant astrocyte hemichannel activity in Juvenile Neuronal Ceroid Lipofuscinosis (JNCL). PLoS ONE. 2014;9:e95023 pubmed publisher
  22. Chen J, Chen J, Zhu Y, Liang C, Zhao H. Deafness induced by Connexin 26 (GJB2) deficiency is not determined by endocochlear potential (EP) reduction but is associated with cochlear developmental disorders. Biochem Biophys Res Commun. 2014;448:28-32 pubmed publisher
  23. Bosen F, Schütz M, Beinhauer A, Strenzke N, Franz T, Willecke K. The Clouston syndrome mutation connexin30 A88V leads to hyperproliferation of sebaceous glands and hearing impairments in mice. FEBS Lett. 2014;588:1795-801 pubmed publisher
  24. Pannasch U, Freche D, Dallérac G, Ghezali G, Escartin C, Ezan P, et al. Connexin 30 sets synaptic strength by controlling astroglial synapse invasion. Nat Neurosci. 2014;17:549-58 pubmed publisher
  25. Holzer L, Cor A, Holzer G. Expression of gap junction proteins connexins 26, 30, and 43 in Dupuytren's disease. Acta Orthop. 2014;85:97-101 pubmed publisher
  26. Xiao J, Zhang G, Qiu P, Liu X, Wu Y, Du B, et al. Tanshinone IIA increases the bystander effect of herpes simplex virus thymidine kinase/ganciclovir gene therapy via enhanced gap junctional intercellular communication. PLoS ONE. 2013;8:e67662 pubmed publisher
  27. Kozoriz M, Lai S, Vega J, Saez J, Sin W, Bechberger J, et al. Cerebral ischemic injury is enhanced in a model of oculodentodigital dysplasia. Neuropharmacology. 2013;75:549-56 pubmed publisher
  28. Zhu Y, Liang C, Chen J, Zong L, Chen G, Zhao H. Active cochlear amplification is dependent on supporting cell gap junctions. Nat Commun. 2013;4:1786 pubmed publisher
  29. Davis N, Phillips A, Becker D. Connexin dynamics in the privileged wound healing of the buccal mucosa. Wound Repair Regen. 2013;21:571-8 pubmed publisher
  30. Forge A, Jagger D, Kelly J, Taylor R. Connexin30-mediated intercellular communication plays an essential role in epithelial repair in the cochlea. J Cell Sci. 2013;126:1703-12 pubmed publisher
  31. Wan C, Shaikh S, Green C, Nicholson L. Comparison of bidirectional and bicistronic inducible systems for coexpression of connexin genes and fluorescent reporters. Anal Biochem. 2012;431:90-5 pubmed publisher
  32. Ghule V, Gray C, Galimberti A, Anumba D. Prostaglandin-induced cervical remodelling in humans in the first trimester is associated with increased expression of specific tight junction, but not gap junction proteins. J Transl Med. 2012;10:40 pubmed publisher
  33. Lynn B, Tress O, May D, Willecke K, Nagy J. Ablation of connexin30 in transgenic mice alters expression patterns of connexin26 and connexin32 in glial cells and leptomeninges. Eur J Neurosci. 2011;34:1783-93 pubmed publisher
  34. Crispino G, Di Pasquale G, Scimemi P, Rodriguez L, Galindo Ramirez F, De Siati R, et al. BAAV mediated GJB2 gene transfer restores gap junction coupling in cochlear organotypic cultures from deaf Cx26Sox10Cre mice. PLoS ONE. 2011;6:e23279 pubmed publisher
  35. Degen J, Schütz M, Dicke N, Strenzke N, Jokwitz M, Moser T, et al. Connexin32 can restore hearing in connexin26 deficient mice. Eur J Cell Biol. 2011;90:817-24 pubmed publisher
  36. Ball K, Harik L, Gandhi G, Cruz N, Dienel G. Reduced gap junctional communication among astrocytes in experimental diabetes: contributions of altered connexin protein levels and oxidative-nitrosative modifications. J Neurosci Res. 2011;89:2052-67 pubmed publisher
  37. Karpuk N, Burkovetskaya M, Fritz T, Angle A, Kielian T. Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity. J Neurosci. 2011;31:414-25 pubmed publisher
  38. Magnotti L, Goodenough D, Paul D. Functional heterotypic interactions between astrocyte and oligodendrocyte connexins. Glia. 2011;59:26-34 pubmed publisher
  39. Schütz M, Auth T, Gehrt A, Bosen F, Körber I, Strenzke N, et al. The connexin26 S17F mouse mutant represents a model for the human hereditary keratitis-ichthyosis-deafness syndrome. Hum Mol Genet. 2011;20:28-39 pubmed publisher
  40. Schütz M, Scimemi P, Majumder P, De Siati R, Crispino G, Rodriguez L, et al. The human deafness-associated connexin 30 T5M mutation causes mild hearing loss and reduces biochemical coupling among cochlear non-sensory cells in knock-in mice. Hum Mol Genet. 2010;19:4759-73 pubmed publisher
  41. Mei X, Ezan P, Giaume C, Koulakoff A. Astroglial connexin immunoreactivity is specifically altered at ?-amyloid plaques in ?-amyloid precursor protein/presenilin1 mice. Neuroscience. 2010;171:92-105 pubmed publisher
  42. Takahashi D, Vargas J, Wilcox K. Increased coupling and altered glutamate transport currents in astrocytes following kainic-acid-induced status epilepticus. Neurobiol Dis. 2010;40:573-85 pubmed publisher
  43. Gandhi G, Ball K, Cruz N, Dienel G. Hyperglycaemia and diabetes impair gap junctional communication among astrocytes. ASN Neuro. 2010;2:e00030 pubmed publisher
  44. Yum S, Zhang J, Scherer S. Dominant connexin26 mutants associated with human hearing loss have trans-dominant effects on connexin30. Neurobiol Dis. 2010;38:226-36 pubmed publisher
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