Inoue D, Chew G, Liu B, Michel B, Pangallo J, D Avino A, et al. Spliceosomal disruption of the non-canonical BAF complex in cancer. Nature. 2019;574:432-436 pubmed publisher

Kelso T, Porter D, Amaral M, Shokhirev M, Benner C, Hargreaves D. Chromatin accessibility underlies synthetic lethality of SWI/SNF subunits in ARID1A-mutant cancers. elife. 2017;6: pubmed publisher

Xiong J, Todorova D, Su N, Kim J, Lee P, Shen Z, et al. Stemness factor Sall4 is required for DNA damage response in embryonic stem cells. J Cell Biol. 2015;208:513-20 pubmed publisher

Li J, Xu J, Jiang H, Zhang T, Ramakrishnan A, Shen L, et al. Chromatin Remodelers Interact with Eya1 and Six2 to Target Enhancers to Control Nephron Progenitor Cell Maintenance. J Am Soc Nephrol. 2021;32:2815-2833 pubmed publisher

Hong A, Tseng Y, Wala J, Kim W, Kynnap B, Doshi M, et al. Renal medullary carcinomas depend upon SMARCB1 loss and are sensitive to proteasome inhibition. elife. 2019;8: pubmed publisher

Gatchalian J, Malik S, Ho J, Lee D, Kelso T, Shokhirev M, et al. A non-canonical BRD9-containing BAF chromatin remodeling complex regulates naive pluripotency in mouse embryonic stem cells. Nat Commun. 2018;9:5139 pubmed publisher

Zhang H, Pandey S, Travers M, Sun H, Morton G, Madzo J, et al. Targeting CDK9 Reactivates Epigenetically Silenced Genes in Cancer. Cell. 2018;175:1244-1258.e26 pubmed publisher

Song J, Saha S, Gokulrangan G, Tesar P, Ewing R. DNA and chromatin modification networks distinguish stem cell pluripotent ground states. Mol Cell Proteomics. 2012;11:1036-47 pubmed