Mouse Anti-Human Amyloid beta, aa 1-17 Monoclonal Antibody, Unconjugated, Clone 6E10 | EMD Millipore MAB1560 product information

This webpage contains legacy information. The product is either no longer available from the supplier or has been delisted at Labome.

product summary

company name :

EMD Millipore

other brands :

Oncogene Research Products, Calbiochem, Novagen, Merck, Upstate Biotechnology, Chemicon, LINCO, Novabiochem, Guava

product type :

antibody

product name :

Mouse Anti-Human Amyloid beta, aa 1-17 Monoclonal Antibody, Unconjugated, Clone 6E10

catalog :

MAB1560

clonality :

monoclonal

host :

mouse

conjugate :

nonconjugated

reactivity :

human, mouse

application :

western blot, immunohistochemistry, immunohistochemistry - frozen section

citations: 12

Published Application/Species/Sample/Dilution	Reference
western blot; mouse; 1:500; fig 5	Manassero G, Guglielmotto M, Zamfir R, Borghi R, Colombo L, Salmona M, et al. Beta-amyloid 1-42 monomers, but not oligomers, produce PHF-like conformation of Tau protein. Aging Cell. 2016;15:914-23 pubmed publisher
immunohistochemistry; mouse; fig 5	Wang J, Lu R, Yang J, Li H, He Z, Jing N, et al. TRPC6 specifically interacts with APP to inhibit its cleavage by Î³-secretase and reduce AÎ² production. Nat Commun. 2015;6:8876 pubmed publisher
immunohistochemistry - frozen section; mouse; 1:2000; fig 1	He P, Staufenbiel M, Li R, Shen Y. Deficiency of patched 1-induced Gli1 signal transduction results in astrogenesis in Swedish mutated APP transgenic mice. Hum Mol Genet. 2014;23:6512-27 pubmed publisher
western blot; human; 1:1000; fig 1	Kukreja L, Kujoth G, Prolla T, Van Leuven F, Vassar R. Increased mtDNA mutations with aging promotes amyloid accumulation and brain atrophy in the APP/Ld transgenic mouse model of Alzheimer's disease. Mol Neurodegener. 2014;9:16 pubmed publisher
	Meng S, Wang B, Li W. Intermittent hypoxia improves cognition and reduces anxiety-related behavior in APP/PS1 mice. Brain Behav. 2020;10:e01513 pubmed publisher
	Velázquez R, Ferreira E, Knowles S, Fux C, Rodin A, Winslow W, et al. Lifelong choline supplementation ameliorates Alzheimer's disease pathology and associated cognitive deficits by attenuating microglia activation. Aging Cell. 2019;18:e13037 pubmed publisher
	Nygaard H, Kaufman A, Sekine Konno T, Huh L, Going H, Feldman S, et al. Brivaracetam, but not ethosuximide, reverses memory impairments in an Alzheimer's disease mouse model. Alzheimers Res Ther. 2015;7:25 pubmed publisher
	Garza Manero S, Arias C, BermÃºdez Rattoni F, Vaca L, Zepeda A. Identification of age- and disease-related alterations in circulating miRNAs in a mouse model of Alzheimer's disease. Front Cell Neurosci. 2015;9:53 pubmed publisher
	He P, Li P, Hua Q, Liu Y, Staufenbiel M, Li R, et al. Chronic administration of anti-stroke herbal medicine TongLuoJiuNao reduces amyloidogenic processing of amyloid precursor protein in a mouse model of Alzheimer's disease. PLoS ONE. 2013;8:e58181 pubmed publisher
	He P, Cheng X, Staufenbiel M, Li R, Shen Y. Long-term treatment of thalidomide ameliorates amyloid-like pathology through inhibition of ?-secretase in a mouse model of Alzheimer's disease. PLoS ONE. 2013;8:e55091 pubmed publisher
	Eimer W, Vassar R. Neuron loss in the 5XFAD mouse model of Alzheimer's disease correlates with intraneuronal A?42 accumulation and Caspase-3 activation. Mol Neurodegener. 2013;8:2 pubmed publisher
	Lawlor P, Bland R, Das P, Price R, Holloway V, Smithson L, et al. Novel rat Alzheimer's disease models based on AAV-mediated gene transfer to selectively increase hippocampal Abeta levels. Mol Neurodegener. 2007;2:11 pubmed