product summary
company name :
Cell Signaling Technology
product type :
antibody
product name :
p57 Kip2 Antibody
catalog :
2557
clonality :
polyclonal
host :
rabbit
conjugate :
nonconjugated
reactivity :
human, mouse
application :
western blot, immunohistochemistry, immunocytochemistry, immunoprecipitation
citations: 20
Published Application/Species/Sample/DilutionReference
  • immunohistochemistry; mouse; 1:500; loading ...; fig s11b
Naito M, Mori M, Inagawa M, Miyata K, Hashimoto N, Tanaka S, et al. Dnmt3a Regulates Proliferation of Muscle Satellite Cells via p57Kip2. PLoS Genet. 2016;12:e1006167 pubmed publisher
  • western blot; human; 1:1000; fig s11e
Shao Z, Zhang R, Khodadadi Jamayran A, Chen B, Crowley M, Festok M, et al. The acetyllysine reader BRD3R promotes human nuclear reprogramming and regulates mitosis. Nat Commun. 2016;7:10869 pubmed publisher
  • western blot; human
Howlin J, Cirenajwis H, Lettiero B, Staaf J, Lauss M, Saal L, et al. Loss of CITED1, an MITF regulator, drives a phenotype switch in vitro and can predict clinical outcome in primary melanoma tumours. Peerj. 2015;3:e788 pubmed publisher
  • immunocytochemistry; human; fig s9e
  • western blot; human; fig s9d
Wang P, Alvarez Perez J, Felsenfeld D, Liu H, Sivendran S, Bender A, et al. A high-throughput chemical screen reveals that harmine-mediated inhibition of DYRK1A increases human pancreatic beta cell replication. Nat Med. 2015;21:383-8 pubmed publisher
Shelby S, Veatch S, Holowka D, Baird B. Functional nanoscale coupling of Lyn kinase with IgE-Fc?RI is restricted by the actin cytoskeleton in early antigen-stimulated signaling. Mol Biol Cell. 2016;27:3645-3658 pubmed
Maggi E, Trillo Tinoco J, Struckhoff A, Vijayaraghavan J, del Valle L, Crabtree J. Retinoblastoma-binding protein 2 (RBP2) is frequently expressed in neuroendocrine tumors and promotes the neoplastic phenotype. Oncogenesis. 2016;5:e257 pubmed publisher
Moreno Lorenzana D, Avilés Vazquez S, Sandoval Esquivel M, Alvarado Moreno A, Ortiz Navarrete V, Torres Martínez H, et al. CDKIs p18(INK4c) and p57(Kip2) are involved in quiescence of CML leukemic stem cells after treatment with TKI. Cell Cycle. 2016;15:1276-87 pubmed publisher
Syu J, Chi J, Kung H. Nrf2 is the key to chemotherapy resistance in MCF7 breast cancer cells under hypoxia. Oncotarget. 2016;7:14659-72 pubmed publisher
Aiken C, Tarry Adkins J, Penfold N, Dearden L, Ozanne S. Decreased ovarian reserve, dysregulation of mitochondrial biogenesis, and increased lipid peroxidation in female mouse offspring exposed to an obesogenic maternal diet. FASEB J. 2016;30:1548-56 pubmed publisher
Sánchez A, Calpena A, Clares B. Evaluating the Oxidative Stress in Inflammation: Role of Melatonin. Int J Mol Sci. 2015;16:16981-7004 pubmed publisher
Gęgotek A, Skrzydlewska E. The role of transcription factor Nrf2 in skin cells metabolism. Arch Dermatol Res. 2015;307:385-96 pubmed publisher
Liu W, Xu C, Wan H, Liu C, Wen C, Lu H, et al. MicroRNA-206 overexpression promotes apoptosis, induces cell cycle arrest and inhibits the migration of human hepatocellular carcinoma HepG2 cells. Int J Mol Med. 2014;34:420-8 pubmed publisher
Panngom K, Lee S, Park D, Sim G, Kim Y, Uhm H, et al. Non-thermal plasma treatment diminishes fungal viability and up-regulates resistance genes in a plant host. PLoS ONE. 2014;9:e99300 pubmed publisher
Hamajima N, Johmura Y, Suzuki S, Nakanishi M, Saitoh S. Increased protein stability of CDKN1C causes a gain-of-function phenotype in patients with IMAGe syndrome. PLoS ONE. 2013;8:e75137 pubmed publisher
Rong M, Chen G, Dang Y. Increased miR-221 expression in hepatocellular carcinoma tissues and its role in enhancing cell growth and inhibiting apoptosis in vitro. BMC Cancer. 2013;13:21 pubmed publisher
Xu L, Wen Z, Zhou Y, Liu Z, Li Q, Fei G, et al. MicroRNA-7-regulated TLR9 signaling-enhanced growth and metastatic potential of human lung cancer cells by altering the phosphoinositide-3-kinase, regulatory subunit 3/Akt pathway. Mol Biol Cell. 2013;24:42-55 pubmed publisher
Hayley M, Bourbigot S, Booth V. Self-association of an activating natural killer cell receptor, KIR2DS1. PLoS ONE. 2011;6:e23052 pubmed publisher
Patel L, Sun W, Glasson S, Morris E, Flannery C, Chockalingam P. Tenascin-C induces inflammatory mediators and matrix degradation in osteoarthritic cartilage. BMC Musculoskelet Disord. 2011;12:164 pubmed publisher
Negrotto S, Hu Z, Alcazar O, Ng K, Triozzi P, Lindner D, et al. Noncytotoxic differentiation treatment of renal cell cancer. Cancer Res. 2011;71:1431-41 pubmed publisher
Farkas B, Boldizsar F, Tarjanyi O, Laszlo A, Lin S, Hutas G, et al. BALB/c mice genetically susceptible to proteoglycan-induced arthritis and spondylitis show colony-dependent differences in disease penetrance. Arthritis Res Ther. 2009;11:R21 pubmed publisher
product information
SKU :
2557P
Product-Name :
p57 Kip2 Antibody
Size :
40 ul
Price-(USD) :
123 USD
Species-x-Reactivity :
H
Applications :
Immunofluorescence (Immunocytochemistry)
Product-Category :
Cell Cycle / Checkpoint Control
Shipping-Temp :
AMBIENT
Storage-Temp :
-20°C
Product-Type :
Polyclonal Antibody
MW :
57
Host :
Rabbit
Target :
p57 Kip2
Primary-Protein :
p57Kip2
Alt-Names :
BWCR,BWS,CDKN1C,CDN1C,Cyclin-dependent kinase inhibitor 1C,Cyclin-dependent kinase inhibitor p57,KIP2,WBS,cyclin-dependent kinase inhibitor 1C (p57, Kip2),p57,p57Kip2
company information
Cell Signaling Technology
3 Trask Lane
Danvers, MA 01923
info@cellsignal.com
www.cellsignal.com
8776162355
headquarters: USA
Established in Beverly, MA in 1999, Cell Signaling Technology (CST) is a privately-owned company with over 400 employees worldwide. We are dedicated to providing innovative research tools that are used to help define mechanisms underlying cell function and disease. Since its inception, CST has become the world leader in the production of the highest quality activation-state and total protein antibodies utilized to expand knowledge of cell signaling pathways. Our mission is to deliver the world's highest quality research tools that accelerate progress in biological research and personalized medicine.