Anti-IL1 beta Rabbit Monoclonal Antibody | Boster M00101-1 product information

This webpage contains legacy information. The product is either no longer available from the supplier or has been delisted at Labome.

product summary

company name :

Boster

product type :

antibody

product name :

Anti-IL1 beta Rabbit Monoclonal Antibody

catalog :

M00101-1

quantity :

100 µl

clonality :

monoclonal

host :

domestic rabbit

conjugate :

nonconjugated

clone name :

BII-9

reactivity :

human

application :

western blot

product information

SKU :

M00101-1

Product Name :

Anti-IL1 beta Rabbit Monoclonal Antibody

Size :

100 µl

Clonality :

Monoclonal

Clone Number :

BII-9

Host :

Rabbit

Reactivity :

Human

Application(s) :

Application Details :

WB 1:500-1:2000

Description :

Boster Bio Anti-IL1 beta Rabbit Monoclonal Antibody catalog # M00101-1. Tested in WB applications. This antibody reacts with Human.

Concentration :

Actual concentration vary by lot. Use suggested dilution ratio to decide dilution procedure.

Gene Name :

IL1B

Uniprot ID :

P01584

Immunogen :

A synthesized peptide derived from human IL1 beta

Form :

Liquid

Contents :

Rabbit IgG in phosphate buffered saline, pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol, 0.4-0.5mg/ml BSA.

Purification :

Affinity-chromatography

Storage :

Store at -20°C for one year. For short term storage and frequent use, store at 4°C for up to one month. Avoid repeated freeze-thaw cycles.

Gene Full Name :

Interleukin-1 beta

Synonyms :

Interleukin-1 beta; IL-1 beta; Catabolin; IL1B; IL1F2;

Molecular Weight :

30748 MW

Protein Function :

Produced by activated macrophages, IL-1 stimulates thymocyte proliferation by inducing IL-2 release, B-cell maturation and proliferation, and fibroblast growth factor activity. IL-1 proteins are involved in the inflammatory response, being identified as endogenous pyrogens, and are reported to stimulate the release of prostaglandin and collagenase from synovial cells.

Subcellular Localization :

Secreted. The lack of a specific hydrophobic segment in the precursor sequence suggests that IL-1 is released by damaged cells or is secreted by a mechanism differing from that used for other secretory proteins.

Research Category :

Atherosclerosis, Cardiovascular, Cytokines, Immunology, Innate Immunity, Interleukins, Metabolism, Obesity, Vascular Inflammation