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- Genentech IL-34 kit was used to perform ELISA in order to show that lysosome function and macrophage homeostasis were disrupted without ENT3.
- Genentech AVPW antagonist was used to perform cell stimulation in order to show that autoubiquitination could be promoted by conformational change of cIAP1 induced by antagonists.
- Genentech BV6 antagonist was used to perform cell stimulation in order to show that autoubiquitination could be promoted by conformational change of cIAP1 induced by antagonists.
- Genentech MV1 antagonist was used to perform cell stimulation in order to show that autoubiquitination could be promoted by conformational change of cIAP1 induced by antagonists.
- Genentech anti-DR5 antibody was used in western blot in order to investigate the effect of death-receptor activation on clathrin-dependent endocytosis.
- Genentech rabbit anti-Shh antibody was used to carry out immunohistochemistry assays in order to determine the regulation of Hedgehog pathway during pancreatic carcinogenesis.
- Genentech mouse monoclonal anti-EGFR antibody was used to carry out immunofluorescence assays in order to investigate the roles for FCHo1 and FCHo2 proteins in clathrin-mediated endocytosis in cell plasma membrane.
- Genentech monoclonal anti-HER2 antibody Herceptin was used as contorl antibody in antibody blocking experiments to investigate the role of PI3k/Akt pathway in macrophage-mediated antibody-dependent cell-mediated cytotoxicity.
- Genentech monoclonal anti-CD20 antibody Rituximab was used to block the cell surface antigen CD20 in Raji cells in order to investigate the role of PI3k/Akt pathway in macrophage-mediated antibody-dependent cell-mediated cytotoxicity.
- Genentech erlotinib was used to treat cells in order to study the synergistic effect of green tea (-) epigallocatechin-3-gallate and EGFR tyrosine kinase inhibitor on inhibition of head and neck tumor growth.
- Genentech anti-HER2 murine monoclonal antibody 4D5 and humanized monoclonal antibody trastuzumab (Herceptin) were used in blocking expriments to study gene regulation by anti-HER2 antibody
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