sample review
  • Alomone anti-NaV1.6 was used to perform immunohistochemistry in order to study the mechanism of the action potential initiation in neocortical inhibitory interneurons.
  • Alomone anti-NaV1.2 was used to perform immunohistochemistry in order to study the mechanism of the action potential initiation in neocortical inhibitory interneurons.
  • Alomone Labs anti-Pan-NaV was used to perform immunohistochemistry in order to study the mechanism of the action potential initiation in neocortical inhibitory interneurons.
  • Alamone Labs rabbit anti-GluR3 antibody was used to perform immunocytochemistry in order to investigate the role of neurotransmitter-mediated exosome transfer in the interactions between neurons and oligodendrocytes.
  • Alomone Labs rabbit polyclonal anti-Nav1.6 was used to perform immunohistochemistry in order to show that brain inhibitory network could be modulated by dendritic gap junctions.
  • Alomone rabbit polyclonal anti-Kv1.2 antibody was used to perform immunohistochemistry and western blot in order to show that TAG1 expression in glial cells was essential for axonal domain differentiation and organization.
  • Alomone labs rabbit anti-GABAA receptor gamma 2 antibody was used to perform immunocytochemistry and ELISA in order to show that NSF and PKC epsilon could regulate GABAA receptor trafficking to affect synaptic inhibition.
  • Alomone rabbit anti-Cav1.2 antibody(1:50) was used to perform immunohistochemistry and western blot in order to study the BIN1 localized Cav1.2 to cardiac T-tubulesbeta
  • Alomone Laboratories rabbit anti-prestin(1:3000) antibody was used to perform immunofluorescence in order to study the critical regulation effect of Eps8 on coordination of development and functionality of mammalian auditory hair cells.
  • Alomone Labs anti-ASIC1 antibody was used to perform western blot in order to study its role in glioblastoma cell migration and whole cell current.
  • Alomone Labs anti-Ca2+ channel alpha 1c antibody was used in western blot in order to investigate the cellular and molecular basis of heart failure (HF) caused by aberrant Ca2+ handling.
sample product